Abstract
Celiac disease (CD) is characterized by a chronicinflammatory state induced by an increased immunologic response to ingested gluten in genet-ically predisposed individuals. Clinical manifestations of CD are multiple and highly variable. In addition to this variability, the belief that CD is arare disease has lead to important under diagnosis. Hematological findings are common, andanemia represents one of its principal manifestations. This review focuses on the pathophysiological mechanisms involved in the genesis ofanemia in CD.
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